Title | Does reversible cysteine oxidation link the Western diet to cardiac dysfunction? |
Publication Type | Journal Article |
Year of Publication | 2014 |
Authors | Behring JB, Kumar V, Whelan SA, Chauhan P, Siwik DA, Costello CE, Colucci WS, Cohen RA, McComb ME, Bachschmid MM |
Journal | FASEB J |
Volume | 28 |
Issue | 5 |
Pagination | 1975-87 |
Date Published | 2014 May |
ISSN | 1530-6860 |
Keywords | Animals, Chromatography, Liquid, Citric Acid Cycle, Cysteine, Diet, Fatty Acids, Glycolysis, Heart Diseases, Male, Mice, Mice, Inbred C57BL, Myocardial Contraction, Myocardium, Obesity, Oxidative Phosphorylation, Oxygen, Phenotype, Protein Processing, Post-Translational, Proteomics, Reactive Nitrogen Species, Reactive Oxygen Species, Sulfhydryl Compounds, Tandem Mass Spectrometry |
Abstract | Using a novel cysteine thiol labeling strategy coupled with mass spectrometric analysis, we identified and quantified the changes in global reversible cysteine oxidation of proteins in the left ventricle of hearts from mice with metabolic syndrome-associated diastolic dysfunction. This phenotype was induced by feeding a high-fat, high-sucrose, type-2 diabetogenic diet to C57BL/6J mice for 8 mo. The extent of reversible thiol oxidation in relationship to the total available (free and reducible) level of each cysteine could be confidently determined for 173 proteins, of which 98 contained cysteines differentially modified ≥1.5-fold by the diet. Our findings suggest that the metabolic syndrome leads to potentially deleterious changes in the oxidative modification of metabolically active proteins. These alterations may adversely regulate energy substrate flux through glycolysis, β-oxidation, citric acid (TCA) cycle, and oxidative phosphorylation (oxphos), thereby contributing to maladaptive tissue remodeling that is associated with, and possibly contributing to, diastolic left ventricular dysfunction. |
DOI | 10.1096/fj.13-233445 |
Alternate Journal | FASEB J. |
PubMed ID | 24469991 |
PubMed Central ID | PMC4046179 |
Grant List | N01 HV028178 / HV / NHLBI NIH HHS / United States HL-064750 / HL / NHLBI NIH HHS / United States R01 HL064750 / HL / NHLBI NIH HHS / United States HHSN268201000031C / HL / NHLBI NIH HHS / United States P01-HL-068758 / HL / NHLBI NIH HHS / United States R01 HL031607 / HL / NHLBI NIH HHS / United States HL-31607 / HL / NHLBI NIH HHS / United States HHSN268201000031C / / PHS HHS / United States P01 HL068758 / HL / NHLBI NIH HHS / United States R37-HL-104017 / HL / NHLBI NIH HHS / United States T32 HL007501 / HL / NHLBI NIH HHS / United States P41 GM104603 / GM / NIGMS NIH HHS / United States R37 HL104017 / HL / NHLBI NIH HHS / United States |
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